[Firestarter]

One of the known causes of immune deficiency are pesticides.
The information in this post shows that DDT and Kelthane made the polio virus much more virulent. It is reasonable to that they would also increase the replication of other viruses.
Technically speaking this isn't "immune deficiency" but the effects would look the same.


In 1972, in the US DDT was prohibited, but instead they continued to produce “dicofol” (a.k.a. Kelthane), which is really DDT in disguise.

DDT was/is used in the United States to control insects in crops and livestock and to combat insect-borne diseases. It was introduced as a pesticide during WWII. In the United States, the general use moratorium took place in 1972, but there is another pesticide dicofol, which is made and sold by Dow AgroSciences and carries the trademark KELTHANE® , is in fact DDT. In China dicofol is produced by the Yangzhou Pesticide Factory, which reports production quantities of 4 million pounds of dicofol per year.
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This class of insecticide (organochlorine) impacts the electrical activity in the body, so it affects brain and heart, but also there are other organs that use electrical current, including the lungs. It clearly affects the immune system and causes cancer, and the best part is that it doesn’t break down in the environment other than to become DDE (and DDD).

K. Paul Stoller – AD, AFP, ALS, and DDT (2015): http://journalijcar.org/sites/default/fi...s/0151.pdf
(archived here: http://archive.is/QmJa7)



Stoller refers to a paper describing research by the US Army Medical Research and Development Command (with MIT) that shows that at the 20 and 40 μg levels of DDT, the yield of polio virus per cell was increased 37 and 90%, respectively.
Also look for Kelthane (that replaced DDT) which increases the yield of the polio virus with a whopping 430%...

insecticidal compounds DDT, chlordane, Kelthane , Dipterex malathion, and Karathane at subtoxic concentrations inhibited vaccinia virus replication in human Chang liver cellq. Under the same experimental conditions, the replication of poliovirus was inhibited only by chlordane and malthion, whereas Kelthane increased the virus yields 4 and 18 times, respectivaly, and DDT exhibited a slight stimulatory effect.
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The studies described in the Annual Progress Report of 1965 indicated that human HeLa cells exposed to subtoxic concentrations of the insecticidal compounds, CygonR, DipterexR, DI-SystonR, chlordane and Karathane for 84 days were more susceptible to poliovirus infection than the corresponding control cells (1,2).
As it is recognized that residues of some agricultural insecticides persist in the body and that cells are continuously exposed to their metabolites, we investigated the possibility that these chemicals may alter certain physiological activities of cells and subsequently influence the susceptibility of hosts to virus infections.
The effects of organophosphorus, organochlorine and dinitrophenol insecticidal compounds on the replication of polio and of vaccinia viruses were studied in human Chang liver cells, using purified or analytical grade comnounds obtained from their manufacturers.
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In the poliovirus test the cell response was not uniform. In comparison to the controls, the virus yield in the DDT-treated cultures was slightly increased; the yield in the chlordane and malathion cultures was reduced (32 and 18% of the controls); and the yield in the Kelthane and Karathane cultures was greatly increased (430 and 1800% respectively).
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The stimulatorv effect by DDT, Kelthane, and Karathane on poliovirus replication suggests a possibility that these compounds may have some specific effects on the mechanisms of viral biosynthesis. This possibility is supported by the results of our previous report which indicated an increased yield of poliovirus in HeLa cells exposed to Karathane for 77 days(2).
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Appendix Number 1
EFFECTS OF INSECTICIDAL COMPOUNDS ON THE REPLICATION OF JACCINIA AND POLIO VIRUSES IN HUMAN CHANG LIVER CELLS
Janis Gabliks
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To investiqate the effects of insecticidal comuounds on viral virus replication, we tested six insecticidal compounds in human liver cells infected with vaccinia and poliovirus.
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In the presence of 20 and 40 μg levels of DDT, poliovirus yield per culture was comparable to that of the control. However, when the yield of infectious virus released is expressed per individual cell or per ig of cell protein, as shown in Figure 3, it is evident that at the 20 and 40 μg levels of DDT, the yield of virus per cell was increased 37 and 90%, respectively. Similarly, the yield per μg of protein was also increased 15 and 47%, respectively
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In contrast to the inhibitory action of these two insecticides, the Kelthane-treated cultures produced about four times more virus, and the Karathane-treated cells 18 times more virus than the corresponding controls.
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The stimulatory effect by DDT, Kelthane, and Karathane on poliovirus replication suggests a possibility that these compounds may have some specific effects on the mechanisms of viral biosynthesis, and the state of some latent viruses may also be altered. This possibility is supported by the results of our previous report which indicated an increased yield of poliovirus in HeLa cells, chronically exposed to Karathane for 77 days (7,8).

J. Gabliks – Studies of Biologically Active Agents in Cells and Tissue Cultures (1967): http://www.dtic.mil/dtic/tr/fulltext/u2/804387.pdf
(archived here: http://archive.is/b7CDg)